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4,7 The link between hearing loss and tinnitus has been suggested to be due to a compensatory effect: the brain compensates for decreased activation of the auditory nerve (which carries signals from the cochlea to the brain) by increasing “gain” or signal amplification in the auditory system of the brain, as well as decreasing auditory signal inhibition, which may result in tinnitus and possibly hypersensitivity to noise

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Due to their extensive involvement in health and disease including cardiovascular disorders 9,10 , neurodegenerative diseases 11,12 and metabolic syndromes 8,13 , GPCRs have emerged as prominent drug targets, with over 30% of Food and Drug Administration (FDA)-approved drugs acting on GPCRs, demonstrating their therapeutic importance 1,14