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The existing literature on the unmodified AEDG sequence provides a robust foundation for hypothesis generation, but researchers should note the distinction between findings established with the parent peptide and the expected – but not yet confirmed – properties of the N-acetylated, amidated form

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GHK-Cu doesn't directly block DHT (that's finasteride's job), but there's evidence it downregulates androgen receptor expression in follicular cells, potentially blunting the follicle's sensitivity to DHT