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Comment: A few commenters expressed concern regarding the reporting and implementation burden of the proposal by requiring the 2025 version of the High Priority Practices SAFER Guide beginning with the CY 2026 performance period/2028 MIPS payment year, particularly for MIPS eligible clinicians with limited resources

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Key Research Areas for FOXO4-DRI FOXO4–p53 protein-protein interaction disruption assays — co-immunoprecipitation (CO-IP) and competitive binding studies Senescent cell identification and selective elimination assays — SA-β-galactosidase, p16, p21, p53 staining combined with FOXO4-DRI apoptosis induction p53 TAD2 binding, nuclear exclusion, and mitochondrial translocation studies — phospho-p53 (Ser15) pathway research BAX/BCL-2/Caspase-3 intrinsic apoptosis pathway investigations in senescent cell models SASP (Senescence-Associated Secretory Phenotype) characterisation and suppression — IL-6, IL-8, MMP, VEGF, CXCL2/3 pathway studies Tissue-specific senescent cell biology — fibroblasts, endothelial cells, Leydig cells, chondrocytes, epithelial cells Vascular ageing pathway research — endothelial cell senescence, aortic function, and ROS/oxidative stress studies Senescent Leydig cell and male reproductive ageing models — FOXO4 nuclear translocation and testosterone synthesis pathway research Chondrocyte senescence and cartilage biology — senescent cell removal and chondrogenic potential restoration studies Keloid and pathological scar biology — senescent fibroblast elimination and SASP suppression in fibrotic tissue models Senescent cancer cell elimination — FOXO4-DRI as a senolytic tool in oncology research models Comparative senolytic research — FOXO4-DRI vs

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