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In this review, we will discuss the possible role of the CYP pathways in alcohol-mediated effects on HIV-1 pathogenesis and HAART medications as follows: i) the effects of alcohol on HIV-1 pathogenesis and neuroAIDS, ii) the role of the CYP pathway in alcohol-mediated oxidative stress and its implications in HIV-1 pathogenesis, iii) the role of the CYP system in alcohol–HAART interactions and its implications in HIV-1 pathogenesis and HAART treatment, iv) conclusions, and v) expert opinion on this subject

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It reduces homocysteine and generates SAMe (S-adenosyl methionine), the most crucial methyl donor we have